How Zika Virus Evades Antiviral Response of Cells

Por um escritor misterioso

Descrição

Researchers have identified a mechanism by which Zika virus can escape detection by host cells, offering a new target for therapeutics.
How Zika Virus Evades Antiviral Response of Cells
Biomedicines, Free Full-Text
How Zika Virus Evades Antiviral Response of Cells
PDF) Zika virus evades interferon-mediated antiviral response through the co-operation of multiple nonstructural proteins in vitro
How Zika Virus Evades Antiviral Response of Cells
Antibody-dependent enhancement - Wikipedia
How Zika Virus Evades Antiviral Response of Cells
An epidemic Zika virus isolate suppresses antiviral immunity by disrupting antigen presentation pathways
How Zika Virus Evades Antiviral Response of Cells
Ploss lab identifies mechanism for Zika virus infection
How Zika Virus Evades Antiviral Response of Cells
Biomedicines, Free Full-Text
How Zika Virus Evades Antiviral Response of Cells
What We're Doing to Combat the Zika Virus University of Washington - Department of Global Health
How Zika Virus Evades Antiviral Response of Cells
Cross-Reactive Dengue Virus Antibodies Augment Zika Virus Infection of Human Placental Macrophages - ScienceDirect
How Zika Virus Evades Antiviral Response of Cells
Zika virus employs the host antiviral RNase L protein to support replication factory assembly
How Zika Virus Evades Antiviral Response of Cells
Constitutive expression and distinct properties of IFN-epsilon protect the female reproductive tract from Zika virus infection
How Zika Virus Evades Antiviral Response of Cells
American-Asian- and African lineages of Zika virus induce differential pro-inflammatory and Interleukin 27-dependent antiviral responses in human monocytes - ScienceDirect
How Zika Virus Evades Antiviral Response of Cells
TLR3 Activation by Zika Virus Stimulates Inflammatory Cytokine Production Which Dampens the Antiviral Response Induced by RIG-I-Like Receptors
How Zika Virus Evades Antiviral Response of Cells
Zika virus antagonizes interferon response in patients and disrupts RIG-I–MAVS interaction through its CARD-TM domains, Cell & Bioscience
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